T. Gondi Toxoplasmosis

Not to be missed in this article is this section is the following section which I find to be fascinating BECAUSE the pseudo-science of psychiatry has few clues as to why most major psychiatric medications work. However, Dr. Howenstein offers science behind the “why”. My hope for the psychiatric community is they would take this type of research seriously in order to FIRST rule out organic causes of mental “illness” rather than prescribing meds based solely on symptoms:

Acute infection with Toxoplasmosis Gondi can produce personality changes and psychosis including delusions and auditory hallucinations. T. Gondii can alter behavior, neurotransmitter function and accounts for approximately 25 % of chorioretinitis usually contracted congenitally. A large study of mentally handicapped persons revealed that the incidence of t.gondii infection in schizophrenic patients was twice that of control subjects*. German research has revealed that first onset schizophrenia patients have a 42 % incidence of antibodies to toxoplasma compared to 11 % in control subjects. T. Gondi usually is spread to humans from cats. Two studies have revealed that exposure to cats in childhood was a risk factor for the development of schizophrenia.

Two of the drugs used to treat psychosis and bipolar disorder (Haldol and Valproic Acid) inhibit the growth of t. gondii in cerebrospinal fluid and blood at concentrations below that being treated with these therapies suggesting that improved mental status might actually be due to killing t. gondii not anti-psychotic effects. The antipsychotic drugs thorazine, haldol and clozapine inhibit viral replication.

Patients with recent onset of schizophrenia have a 400 % increase in reverse transcripyase activity in their cerebrospinal fluid which is seen in patients with infectious retroviruses

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